Disorders Of The Veins And Research Paper

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Mostly proteins and platelets combine to form blood clotting in order to heal wounds inside the arteries. However, the accumulation of fatty protein and platelet deposits on the artery walls can sometimes cause the arteries to become narrow and hardened. This makes the artery more prone to damage and if it ruptures, the blood clotting occurs and thus, the artery becomes infected by arterial thrombosis. Impact of patient age on the path physiology of CVI and DVT

The age factor is critical towards understanding how a disease might impact the sick person and how the person's body would respond to the various treatments applied on it. As mentioned earlier, these disorders are more common in the adult population. In the case of CVI, the vein walls weaken as the age of the person increases. This is due to the reason that the internal system of the person has become old, weak and slow. Consequently, the muscle layers in the veins of an infected area easily breakdown and the growth of CVI is quicker as compared to in a person of young age. Similarly, the DVT is also dependent upon the age of the patient.

Treatment of CVI and DVT...

...

However, in elderly patients, the low weight molecular herparin prescribed so that they can endure its effects. Further development of the disease is the best cure at any age. This can be achieved by regularly exercising the calf muscles, and walking few hours a day. This can lead to improved functioning of the calf. Other factors to monitor would be controlling and maintaining as close to normal blood pressure as possible. Patient diagnosed with CVI are recommended to elevate their feet above their thighs when they are in a sitting position and above their heart, when they are lying down. This exercise should be performed three or four times a day in order to reduce/control the swelling in the calf.
Bibliography

Chieh-Min Fan, M. (2005). Venous Pathophysiology. Semin Intervent Radiol, 22(3), 157 -- 161.

Lopez JA, C.J. (2009). Pathophysiology of venous thrombosis. Thromb Res 123 (suppl 4), S30 -- S34.

Sources Used in Documents:

Bibliography

Chieh-Min Fan, M. (2005). Venous Pathophysiology. Semin Intervent Radiol, 22(3), 157 -- 161.

Lopez JA, C.J. (2009). Pathophysiology of venous thrombosis. Thromb Res 123 (suppl 4), S30 -- S34.


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