- Words: 1101
- Length: 3 Pages
- Topic: Disease
- Paper #: 84221798
This then leads to the activation of a number of genes whose products trigger cell-cycle arrest, apoptosis, or DNA repair" (Lakin 1999, p. 7644).
In research led by Hussain, he investigated the targets of free radicals, which are DNA, proteins, RNA, and lipids. He noted that, "mutations in cancer-related genes or post-translational modifications of proteins by nitration, nitrosation, phosphorylation, acetylation or polyADP-ribosylation-by free radiacals or lipid peroxidation byproducts…are some of